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Kailasa (Clarithromycin)

Kailasa is used in combination with other medicines to eliminate H. pylori - a bacterium that causes ulcers. It is sometimes used to treat other types of infections including Lyme disease, cryptosporidiosis, cat scratch disease, Legionnaires' disease, and pertussis (whooping cough). It is occasionally used to prevent heart infections in people having dental or other procedures. Kailasa is in a class of medications known as macrolide antibiotics. It works by stopping the growth of bacteria. Like all antibiotics, it cannot kill viruses that cause colds, flu or other viral infections.

Other names for this medication:
Clarithromycin, Biaxin, Abbotic, Aeroxina, Biclar, Binoclar, Bremon, Clabel, Clamycin, Clarimac, Clarimax, Clarimed, Clarimycin, Claripen, Claritt, Clariva, Clariwin, Clarix, Claromycin, Claron, Cleron, Emimycin, Euromicina, Finasept, Fromilid, Infex, Kailasa, Kalixocin, Klabax, Klabet, Klabion, Klarimac, Klarimax, Klarithran, Klax, Klaz, Klerimed, Kofron, Krobicin, Lekoklar, Macladin, Maclar, Macrobid, Macrol, Monozeclar, Naxy, Synclar, Taclar, Veclam, Vikrol, Xylar, Zeclar, Zeclaren,

Similar Products:
Glumetza, Zepro, Digitek, Oleptro, Tervalon, Acimed, Omnitan, Herpesil, Cozac, Pansec, Fexidine, Florazole, Flagicure, Selgian, Penrazol, Amoxipenil, Ambilan, Sarvas, Herclov, Tetralisal, Cardivas, Beclabuvir, Myambutol, Padet, Aman, Parox, Metolazone, Peptazol, Zanprol, Norwin,

Kailasa comes as a tablet, an extended-release (long-acting) tablet, and a suspension (liquid) to take by mouth. The regular tablet and liquid are usually taken with or without food every 8 (three times a day) to 12 hours (twice a day) for 7 to 14 days. The extended-release tablet is usually taken with food every 24 hours (once a day) for 7 to 14 days. Your doctor may tell you to take Kailasa for a longer time depending on your condition. Take Kailasa at around the same time(s) every day. Follow the directions on your prescription label carefully, and ask your doctor or pharmacist to explain any part you do not understand. Take Kailasa exactly as directed. Do not take more or less of it or take it more often than prescribed by your doctor.

Shake the suspension well before each use to mix the medication evenly.

Swallow the long-acting tablets whole; do not split, chew, or crush them.

You should begin to feel better during the first few days of treatment with Kailasa. If your symptoms do not improve or get worse, call your doctor.

Take Kailasa until you finish the prescription, even if you feel better. If you stop taking Kailasa too soon, or skip doses, your infection may not be completely treated and the bacteria may become resistant to antibiotics.

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Pelvic organ prolapse: is there a difference in POPQ exam results based on time of day, morning or afternoon? SERENADE: the Study Evaluating Rimonabant Efficacy in Drug-naive Diabetic Patients: effects of monotherapy with rimonabant, the first selective CB1 receptor antagonist, on glycemic control, body weight, and lipid profile in drug-naive type 2 diabetes. Intergenic variants of HBS1L-MYB on chromosome 6q23 have recently been shown to be a major quantitative trait locus (QTL) influencing HbF levels in normal Caucasian adults.

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Endothelial cells and the pathogenesis of lepromatous neuritis:insights from the armadillo model. Reutilization of iron by the spleen reticuloendothelial cells is subjected to inhibition by excess copper.

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Clinical pharmacology of carbohydrate derivatives with cytostatic potency. Reports of animal disease included abortions and deaths in young animals. Comparison of refractive outcome using intraoperative biometry and partial coherence interferometry in silicone oil-filled eyes. Furthermore, for both cell populations, immune suppression of the host has been necessary for long-term retention of transplanted cells. We designed an experiment to compare single-dose versus multidose cardioplegia (calcium 0.3 mmol/L) in neonatal rabbit hearts 1, 4 and 6 weeks of age at 25 degrees C and 32 degrees C.

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Using NT-proBNP as a criterion for heart failure hospitalization. We explored that green LED (530 nm) irradiation directed the human orbital fat stem cells (OFSCs) to migrate away from the LED light source through activation of extracellular signal-regulated kinases (ERK)/MAP kinase/p38 signaling pathway. Chloroplast cytochrome b-563, hydrophobic environment and lack of direct reaction with ferredoxin. Acute hepatitis in the form of severe icterus, successfully treated with ACTH and cortisone

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These findings argue that GluA1-dependent synaptic plasticity might be critically involved in the expression of depression. Extranodal natural killer (NK)/T-cell lymphoma, nasal type, and aggressive NK-cell leukemia are highly aggressive diseases with a poor outcome.

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TNF-alpha-induced NF-kappaB signaling reverses age-related declines in VEGF induction and angiogenic activity in intervertebral disc tissues. Detailed results are given which described three types of language delay at age three: delayed verbal comprehension only, delayed verbal expression only, and delayed development in both aspects of language.

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Neonatal spongioform myelinopathy after restricted application of hexachlorophane skin disinfectant. It is concluded that, at the mineralization front, cell death is linked directly to the elevation in environmental anion concentration and the concomitant rise in intracellular Pi levels. Monkey cerebral arterial relaxation caused by hypercapnic acidosis and hypertonic bicarbonate. Characterization of multiple forms of acetylcholinesterase in electric organ of Torpedo marmorata.

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The affected individuals did not share haplotypes for genetic microsatellite markers surrounding genes that are known to cause autosomal-dominant corneal dystrophies. Both local perfusion pressure and vascular resistance decreased during carotid occlusion. At the time of the reconstruction, high-resolution images are reloaded while forcing a left-right axial flip.

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We aimed to determine the alcohol consumption, blood alcohol levels (BALs) and subsequent driving of patrons leaving 15 hotels and taverns in Perth, Western Australia. Long-term culture of human immunodeficiency virus type 1 resulting in loss of glycosylation sites. The p21-activated kinase is required for neuronal migration in the cerebral cortex.

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Three subjects in the SW group showed abnormalities: one during both the resting record (RR) and hyperventilation (HV), and two only during HV. However, unlike erythropoietin, darbepoietin-alfa did not impair bone marrow B lymphopoiesis. Since the early 70s electrochemistry has been used as a powerful analytical technique for monitoring electroactive species in living organisms. Direct-to-consumer theranostics, 21st century collective innovation and entrepreneurship.

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The article is based on one of the longest established stroke services in Melbourne, Australia. Electron microscopic and serological techniques have been used to study the relationship between cell surface expression of bovine erythrocyte antigens and the genes coding for these antigens.

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A retrospective review of clinical features, microbiological diagnosis, and outcome of community acquired lobar pneumonia was carried out in HIV positive patients admitted to a specialist unit from 1987 to 1993. Allergic sensitization of the skin and oral mucosa to acrylic denture materials. In addition, great attention must be paid to management of the soft tissue tensions and placement of the component parts relative to soft tissue origins and insertions so that asymmetric overloading of the components can be avoided and loosening and wear can be minimized. Gives the objectives and methodology, main findings and recommendations, together with a cost benefit analysis of the work.

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An alternative to petri dishes in quantitative mutation experiments with cultured mammalian cells. A new technique for separation of the CNBr cleavage products of collagen is described. To discuss the technique and clinical applications of myocardial and hepatic H magnetic resonance spectroscopy to study myocardial and hepatic triglyceride content, in relation to changes in plasma nonesterified fatty acids induced by nutritional interventions.

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